Abstract
We introduce Causal Computational Asymmetry (CCA), a principle for causal direction identification based on optimization dynamics in which one neural network is trained to predict Y from X and another to predict X from Y, and the direction that converges faster is inferred to be causal. Under the additive noise model Y=f(X)+ε with ε⊥X and f nonlinear and injective, we establish a formal asymmetry: in the reverse direction, residuals remain statistically dependent on the input regardless of approximation quality, inducing a strictly higher irreducible loss floor and non-separable gradient noise in the optimization dynamics, so that the reverse model requires strictly more gradient steps in expectation to reach any fixed loss threshold; consequently, the forward (causal) direction converges in fewer expected optimization steps. CCA operates in optimization-time space, distinguishing it from methods such as RESIT, IGCI, and SkewScore that rely on statistical independence or distributional asymmetries, and proper z-scoring of both variables is required for valid comparison of convergence rates. On synthetic benchmarks, CCA achieves 26/30 correct causal identifications across six neural architectures, including 30/30 on sine and exponential data-generating processes. We further embed CCA into a broader framework termed Causal Compression Learning (CCL), which integrates graph structure learning, causal information compression, and policy optimization, with all theoretical guarantees formally proved and empirically validated on synthetic datasets.
More in this topicCausal discovery infers cause-and-effect structures from observational and interventional data, distinguishing causal relationships from correlation.
See all 221 papers → Apr 29, 2026
Can Neural Assemblies -- groups of neurons that fire together and strengthen through co-activation -- learn the direction of causal influence between variables? While established as a computationally general substrate for classification, parsing, and planning, neural assemblies have not yet been shown to internalize causal directionality. We demonstrate that the inherent operations of neural assemblies -- projection, local plasticity control, and sparse winner selection -- are sufficient for directional learning. We introduce DIRECT (DIRectional Edge Coupling/Training), a mechanism that co-activates source and target assemblies under an adaptive gain schedule to internalize directed relations. Unlike backpropagation-based methods, DIRECT relies solely on local plasticity, making the resulting causal claims auditable at the mechanism level. Our findings are verified through a dual-readout validation strategy: (i) synaptic-strength asymmetry, measuring the emergent weight gap between forward and reverse links, and (ii) functional propagation overlap, quantifying the reliability of directional signal flow. Across multiple domains, the framework achieves perfect structural recovery under a supervised, known-structure setting. These results establish neural assemblies as an auditable bridge between biologically plausible dynamics and formal causal models, offering an "explainable by design" framework where causal claims are traceable to specific neural winners and synaptic asymmetries.
Evangelia Kopadi, Dimitris Kalles
Hellenic Open University Greece
Mar 27, 2026
Identification of causal directionality in bivariate numerical data is a fundamental research problem with important practical implications. This paper presents two alternative methods to identify direction of causation by considering conditional distributions: (1) Anticipated Asymmetric Geometries (AAG) and (2) Monotonicity Index (MI). The AAG method compares the actual conditional distributions to anticipated ones along two variables. Different comparison metrics, such as Pearson correlation, cosine distance, Hellinger distance, Jaccard index, Jeffreys divergence, K-L divergence, K-S distance, MAE, MSE, mutual information, and Wasserstein distance have been evaluated. Anticipated distributions have been projected as normal based on dual response statistics: mean and standard deviation. The MI method compares the calculated monotonicity indexes of the gradients of conditional distributions along two axes and exhibits count of gradient sign changes. Both methods assume stochastic properties of the bivariate data and exploit anticipated unimodality of conditional distributions of the effect. The proposed methods are straightforward and include only a limited number of hyperparameters that affect the accuracy of the identification. For a given set of hyperparameters, both the AAG and MI methods provide a unique, deterministic solution. To address sensitivity to hyperparameters, tuning has been done by utilizing a full factorial Design of Experiment. It turns out that the AAG method outperforms MI, achieving top weighted accuracies of 82.7% with simple tuning and 84.4% with size-adaptive tuning, compared with 81.6% for GRCI or 82.0% for CAREFL-H on the 99 pairs of the Tubingen real-world cause-effect examples. To evaluate the decisiveness of the identification method, a decision tree was fitted on the input data's symmetrical bivariate statistics to detect misclassified cases.
Alex Glushkovsky
Apr 2, 2026
Understanding causal dependencies in observational data is critical for informing decision-making. These relationships are often modeled as Bayesian Networks (BNs) and Directed Acyclic Graphs (DAGs). Existing methods, such as NOTEARS and DAG-GNN, often face issues with scalability and stability in high-dimensional data, especially when there is a feature-sample imbalance. Here, we show that the denoising score matching objective of diffusion models could smooth the gradients for faster, more stable convergence. We also propose an adaptive k-hop acyclicity constraint that improves runtime over existing solutions that require matrix inversion. We name this framework Denoising Diffusion Causal Discovery (DDCD). Unlike generative diffusion models, DDCD utilizes the reverse denoising process to infer a parameterized causal structure rather than to generate data. We demonstrate the competitive performance of DDCDs on synthetic benchmarking data. We also show that our methods are practically useful by conducting qualitative analyses on two real-world examples. Code is available at this url: https://github.com/haozhu233/ddcd.
Hao Zhu, Di Zhou, Donna Slonim
Beth Israel Deaconess Medical Center Harvard Medical School Boston, MA, USA · Department of Computer Science Tufts University Medford, MA, USA