Beyond Differences: Doubly Robust Meta-Learners for Ratio-Based Treatment Effects

Conditional Average Treatment Effect (CATE) estimation in practice demands three properties simultaneously: heterogeneous effects $τ(x)$, calibrated uncertainty over them, and robustness to the heavy tails that contaminate real outcome data. Meta-learners (Künzel et al., 2019) give (i); causal forests and BART give (i)-(ii) with Gaussian-tail assumptions; no widely used tool gives all three. We present Bayesian X-Learner, an X-Learner built on cross-fitted doubly robust pseudo-outcomes (Kennedy, 2020) with a full MCMC posterior over $τ(x)$ via a Welsch redescending pseudo-likelihood. On Hill's IHDP benchmark the default configuration attains mean $\sqrt{\varepsilon_{\mathrm{PEHE}}} = 0.56$ on 5 replications (lowest mean; differences from S-/T-/X-learners, full-config Causal BART, and a causal forest baseline are not significant at $α=0.05$, and rank ordering is unstable at 10 replications -- IHDP comparisons are competitive rather than dominant). On contaminated "whale" DGPs with up to 20-25% tail density, a one-flag extension (contamination_severity) that selects a Huber-$δ$ nuisance loss per Huber's minimax-$δ$ relation recovers RMSE $\approx 0.13$ with tight credible intervals (single-cross-fit 30-seed coverage 83% [Wilson 66%, 93%] at 20% density; modular-Bayes pooling with Bayesian-bootstrap nuisance draws restores nominal 95% coverage).

Unobserved confounding prevents standard covariate adjustment from identifying causal response functions in observational studies. Proxy causal learning addresses this problem through bridge equations involving treatment- and outcome-inducing proxies, avoiding direct recovery of the latent confounder. Existing doubly robust proxy estimators combine outcome and treatment bridges, but typically rely on fixed kernels, sieves, or low-dimensional semiparametric models; existing neural proxy methods are more flexible, but are largely single-bridge estimators. We develop a neural doubly robust framework for proxy causal learning with continuous and structured treatments. Our method introduces a neural mean-embedding estimator for the treatment bridge, combines it with a neural outcome bridge, and estimates the doubly robust correction through a final regression stage. The framework covers population, heterogeneous, and conditional dose-response functions, yielding full response-curve estimators rather than binary-treatment effects. The algorithms use two stages for each bridge and history-aware updates of the final linear layers to stabilize stochastic multi-stage training. We prove consistency of the algorithms showing that the doubly robust error is controlled by the final averaging and regression errors together with the smaller of the outcome- and treatment-side weak-norm bridge errors. Across synthetic and image-valued benchmarks, the proposed estimators outperform existing baselines and single-bridge neural estimators, showing the benefit of combining learned outcome and treatment bridges in a doubly robust construction. Our implementation is available at https://github.com/BariscanBozkurt/DRPCL-Neural-Mean-Embedding.

Policy learning methods are increasingly used to inform treatment allocation under budget constraints. Most proposed methods assume complete treatment data, yet applications frequently suffer from missingness that can bias estimates and lead to suboptimal policies. We address this gap by extending efficient estimators for average treatment effect (ATE) estimation to policy value and conditional average treatment effect (CATE) estimation under missing at random (MAR) and missing completely conditionally at random (MCCAR) treatment data. Through asymptotic efficiency analysis, we prove that the MAR estimator, which leverages partially-observed units, is both valid and more efficient than the MCCAR estimator when MCCAR assumptions hold. This result provides formal justification for preferring MAR-based estimation in policy learning under both missing data settings. Our comprehensive experiments using synthetic and semi-synthetic datasets confirm that correctly specifying the missingness mechanism is crucial: misspecified estimators remain biased regardless of sample size, while our estimators achieve near-oracle performance when assumptions are satisfied. Our work provides practitioners with theoretically grounded, empirically validated tools for robust policy learning in the presence of missing treatment data.