Transportable Causal Effect Estimation across Networks under Interference

Transporting a causal conclusion from a source study population to a target one is a fundamental problem in causal inference. The theory of transportability provides a criterion for when this is possible: given experimental data from the source and observational data from the target, it determines whether a target query is identifiable and does so completely; i.e. if the query can be transported, the criterion finds the exact formula. However, it works one query at a time and returns an expression rather than the value itself. It is also silent in two practically important regimes: when the query is not transportable and when no target data exist at all. To tackle both, we take a model-level perspective grounded in Causal Abstraction theory. Source and target share variables, graph, and interventions, differing only at a known set of mechanisms, which makes transportability a special case of same-level abstraction. Thus, instead of asking whether one query transports, we ask whether a single map aligns the source and target across their interventional behaviour. We characterise when such a map exists in both the Markovian and semi-Markovian settings; when it does, every target query transports at once. Our main contribution lies in the approximate case. When no exact map exists, the best approximate one still yields certified query intervals, recasting abstraction error as a quantitative notion of approximate transportability. We formulate model-level transport as distributionally robust optimisation over mechanism and environment perturbations of the unseen target and derive certificates for both challenging regimes: bounds for non-transportable queries, and guarantees under target-agnostic settings. We evaluate our framework on synthetic Markovian and semi-Markovian benchmarks and a real ecological dataset, and we show that the certified intervals bracket the true interventional query.

Estimating causal effects under networked interference from observational data is a crucial yet challenging problem. Most existing methods mainly rely on the networked unconfoundedness assumption, which guarantees the identification of networked effects. However, this assumption is often violated due to the latent confounders inherent in observational data, thereby hindering the identification of networked effects. To address this issue, we leverage the rich interaction patterns between units in networks, which provide valuable information for recovering these latent confounders. Building on this insight, we develop a confounder recovery framework that explicitly characterizes three categories of latent confounders in networked settings: those affecting only the unit, those affecting only the unit's neighbors, and those influencing both. Based on this framework, we design a networked effect estimator using identifiable representation learning techniques. From a theoretical standpoint, we prove the identifiability of all three types of latent confounders and, by leveraging the recovered confounders, establish a formal identification result for networked effects. Extensive experiments validate our theoretical findings and demonstrate the effectiveness of the proposed method.

Conformal counterfactual prediction constructs prediction sets with finite-sample coverage guarantees for counterfactual outcomes and individual treatment effects under the no-interference assumption. In this work, we relax this assumption by allowing each unit's potential outcomes to depend on other units' treatments and covariates. In this setting, propensity-score reweighting does not restore weighted exchangeability, and existing methods may fail to achieve valid coverage. To address this issue, we develop interference-adjusted weighted conformal prediction that accounts for interference by constructing an observable upper bound on the ideal and unobserved conformal $p$-value under the target intervention. The resulting prediction sets provide finite-sample marginal coverage guarantees for counterfactual outcomes and individual treatment effects in both transductive and inductive settings. We also derive a sharper construction when intervention-induced changes in nonconformity scores are bounded. Numerical experiments show that our methods preserve nominal coverage, whereas existing methods may not.